“Innovation without access is not an innovation… it is an injustice.”

Discovery of a multicomponent alloy forged by the Hiroshima atomic blast

High-energy nuclear detonations generate extreme, transient physicochemical environments capable of producing previously unknown materials. We report the discovery of a previously unknown multicomponent alloy preserved within a hiroshimaite spherule recovered from beach sands of Hiroshima Bay, formed during the 6 August 1945 atomic airburst. The micrometer-sized metallic grain occurs within a quenched glassy matrix. Electron microprobe analyses reveal a homogeneous, Si-rich multielement composition (Fe-Cr-Ni-Mn-Mo-Si-Al). Single-crystal x-ray diffraction shows that the phase crystallizes in space group P213 with the ordered AlAu4-type structure, an ordered derivative of β-Mn. The alloy likely formed by condensation from a mixed metallic vapor followed by ultrafast quenching in the expanding fireball. This finding demonstrates that nuclear plasma events may stabilize complex metallic phases and highlights atomic-blast debris as a natural laboratory for nonequilibrium alloy formation and materials discovery.

https://www.science.org/doi/10.1126/sciadv.aeg8299

Invasive species emerge as a driver of human displacement, new research shows
Invasive alien species degrade ecosystems, erode food and livelihood security, and act as a direct driver of forced human displacement. Case examples include Opuntia stricta in Kenya, little fire ants in Pacific islands, and water hyacinth in African fisheries. Biological invasions are rarely integrated into migration or sustainability science, and the paper urges their inclusion in international indicators and policy on displacement and land degradation.
The new study argues that biological invasions can erode natural resources vital to human security, forcing communities to abandon their land and livelihoods. Despite these impacts, the link between invasive species and human displacement has received little attention.
Researchers call for stronger international action to address this issue.

Reaser JK and Witt ABR (2026) Invasive alien species are drivers of human displacement: recommendations for a multilateral mitigation framework. Front. Conserv. Sci. 7:1858063. doi: 10.3389/fcosc.2026.1858063

https://www.frontiersin.org/journals/conservation-science/articles/...

Phage and microbiota therapy reduced recurrent UTIs in two patients over two years
Combined phage therapy and fecal microbiota transplantation were administered to three women with antibiotic-refractory recurrent E. coli UTIs. Treatment was well tolerated. Over two years, the two patients receiving both phage therapy and FMT showed sustained reduction of UTIs and antibiotic use, while phage therapy alone led to continued but milder episodes.

https://www.nature.com/articles/s41564-026-02409-0

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Mitochondria: Scientists uncover new role for fighting bacterial infections, including drug-resistant superbugs

Mitochondria: They are the engines of cellular energy production; they possess their own DNA, and they are passed from generation to generation from mothers to offspring.

Now scientists have revealed that these bean-shaped energy factories, known for the production of ATP, the key fuel of life, also play an extraordinary role in harnessing the immune response during infection—these organelles are critical in mediating a powerful antibacterial defense.

The discovery demonstrates an unexpected role for these crucial constituents of cells. The team showed that mitochondrial fission underlies an immune response, producing antibacterial capability in a host of organisms from the lowly worm to complex mammals.

The findings suggest that mitochondrial fission is an evolutionarily conserved pathway that provides cell-autonomous control of infection and can be targeted to combat antibiotic-resistant bacterial pathogens.

Mitochondrial division—or fission—can promote antibacterial defense pathways in mammalian macrophages and worms, according to the study, which identified the regulatory mechanisms that control these defense pathways. Findings from the research could inform future therapeutic targets while highlighting unique mitochondrial functions in the fight against infection.

Part 1

Fission creates more mitochondria As a first step in their experiments, scientists infected mouse macrophages with E. coli and found that the number of mitochondria increased, a direct result of fission. The fission, in this instance, was induced in response to E. coli infection. Then, with the help of a cocktail of compounds and the silencing of a gene involved in fission, scientists wanted to see what would happen if the organelles were directed toward mitochondrial fusion—the opposite pathway of fission. The result was increased intracellular E. coli in the macrophages. Still, the lesson learned from E. coli was not universal, and there apparently are significant differences in mitochondrial immune reactions from one pathogen to another. With Salmonella typhimurium, for example, researchers found that the pathogen survives inside macrophages and orchestrates mitochondrial behavior to the benefit of the bacteria, preventing mitochondrial fission and the substantial increase in additional mitochondria. But researchers had an answer to the craftiness of Salmonella typhimurium: Inhibiting the enzyme HDAC6 promoted mitochondrial fission and lipid droplet formation, enhancing bacterial clearance—and suggesting that HDAC6 could be a therapeutic target to control otherwise recalcitrant infections.

Ronan Kapetanovic et al, Mitochondrial fission mediates an evolutionarily conserved antibacterial defense response, Science Immunology (2026). DOI: 10.1126/sciimmunol.aed2623

Part 2

Bacteria build a mineral shield that could help marine infrastructure resist rust

Researchers have developed a biological method for protecting marine infrastructure from corrosion caused by prolonged exposure to seawater. The study, published in Cell Reports Physical Science, presents an environmentally friendly alternative to the chemical corrosion inhibitors currently used to protect metals—materials that can be both polluting and costly.
The researchers discovered that these bacteria work cooperatively to form a dense, durable and naturally occurring protective layer on the surfaces of metals commonly used in marine and electrochemical infrastructure, including steel and iron.

Experiments conducted as part of the study demonstrated that this protective coating significantly slows the corrosion process that causes metals to rust and deteriorate in seawater. The researchers also found that each bacterial species plays a distinct role: Some create the conditions necessary for the process, while others produce an enzyme that enables the formation of the mineral protective layer.
A seawater-derived bacterial consortium, including Bacillus subtilis, forms a dense mineralized biofilm on steel and iron that markedly slows marine corrosion. Different species fulfill complementary roles in conditioning the surface and producing enzymes that drive mineral layer formation. Multispecies communities provide more robust, long-term protection than single strains, offering a potential low-toxicity alternative to chemical corrosion inhibitors.

Avichay Nahami et al, Synthetic bacterial communities as synergistic anti-corrosion agents, Cell Reports Physical Science (2026). DOI: 10.1016/j.xcrp.2026.103419

Not all exercise is equal when it comes to metabolic syndrome risk
Exercise modalities showed distinct effects on metabolic syndrome markers. Aerobic plus resistance training improved waist circumference, BMI, LDL cholesterol and glycemic control; mind-body exercise improved HDL cholesterol and systolic blood pressure; high- and low-volume HIIT preferentially reduced body fat and lipids or diastolic pressure, respectively. Evidence certainty was generally low.

Xinyu Shen et al, Comparative efficacy of various exercise therapies for metabolic syndrome a systematic review and network meta-analysis, iScience (2026). DOI: 10.1016/j.isci.2026.116801

Leaky gut might make food allergies worse

A "leaky gut" might increase the severity of food allergies, according to new research conducted in mice and published in The Journal of Immunology. The researchers found that changes to the gut microbiome in mice were linked to increased intestinal permeability (leaky gut) and increased food allergy severity.

Intestinal permeability describes how the intestines move water and nutrients from food into the bloodstream. Increased permeability, sometimes called "leaky gut," describes a condition in which damage to the gut lining allows partially digested food, toxins or germs to pass into the bloodstream.

The gut microbiome—the trillions of bacteria living in the gut—directly regulates this barrier, meaning microbiome imbalances can be a primary driver of gut leakage.
In mice, distinct gut microbiomes were associated with differing intestinal permeability, Th2/Treg balance, and food allergy severity. Cohousing transferred microbiota from severely affected mice to mildly affected mice, increasing intestinal permeability and worsening allergic responses. Data indicate gut microbes modulate barrier function and food-allergy–specific immune cells.
In this study, the researchers used two groups of mice known to have differences in their gut microbiomes. When a food allergy was induced in the mice, one group developed mild allergic reactions (less diarrhea), while the other had more severe reactions. The mice with more severe diarrhea were also known to have increased intestinal permeability, or "leaky gut."

In mice with more severe food allergies, the researchers also observed an increase in immune cells that cause allergic reactions, Th2 cells, and a decrease in food-allergy-specific immune cells that turn off immune responses, Treg cells. Th2 cells are the immune cells that cause anaphylaxis in people with food allergies.
Interestingly, when the researchers housed mice from the two groups together, they found that mice with milder food allergies developed more severe symptoms. Further tests uncovered that the gut microbiome of the mice with milder allergic reactions shifted to be more like that of the group with severe reactions. The researchers also reported increased intestinal permeability and a change in the balance of immune cells involved in the food allergy response, suggesting that "leaky gut" and the gut microbiome are linked to how the immune system responds to a food allergy.
Researchers clearly saw that food-allergy-specific immune cells were impacted by gut microbes, which affected the food allergy itself.

Journal of Immunology (2026). DOI: 10.1093/jimmun/vkag172

Overactive Neurons Cause Excessive Sweating

Identification of gene variants that affect the activity of neuronal sodium channels could help develop targeted therapies for patients suffering from excessive sweating.
Sweating is a normal, although sometimes unpleasant, part of being human. For people with primary idiopathic hyperhidrosis (PIH), though, excessive sweating can impede their quality of life. Despite affecting up to five percent of people, though, scientists don’t fully understand the mechanisms behind this overactive salty secretion.

Researchers have found that some patients have alterations to their sweat glands’ morphology or function, possibly contributing to the extra sweating. But for people who sweat excessively despite having normal looking sweat glands, scientists suspect that the cause could be hyperactive sympathetic neurons.
In a study published recently in Science Advances, researchers dug into the genetic sequences of 32 families with confirmed PIH to find possible sources of overactive sweat responses. They identified a lead candidate in SCN10A, a gene encoding a sodium channel in sympathetic neurons and discovered that introducing it into mice reproduced the extra sweating phenotype of PIH individuals. They showed how their mouse model of PIH could be used to explore new treatment options for patients.
For decades, hyperhidrosis has largely been viewed as a disorder of the sweat glands. These new findings show that, for some patients, the problem actually begins in the nerves that control sweating, providing a biological explanation for the condition and a potential pathway to more effective treatments.

1. Wohlrab J, et al. Hyperhidrosis: A central nervous dysfunction of sweat secretion. Dermatol Ther. 2023;13(2):453-463.
2. Yamauchi S, et al. A neurocutaneous NaV1.8 channelopathy underlies a genetic subtype o.... Sci Adv. 2026;12(29):aed3221.

Scientists identify fructose as a surprise driver of cancer spread

A new study has uncovered an unexpected link between fructose—a common dietary sugar—and the spread of an aggressive form of ovarian cancer. Published in Nature Aging, the study found that cancer cells not killed by chemotherapy send signals to neighboring tumor cells, helping them become more capable of spreading.

The researchers identified fructose as a key messenger in this process, revealing a previously unrecognized way that treatment-surviving cancer cells may promote the spread of cancer.
Even more interesting, they found that without chemotherapy treatment, consuming the high levels of fructose found in sugary drinks can also signal cancer cells to spread. The fructose finding is especially compelling because it raises the possibility that simple dietary changes could help shape how cancer progresses.

Chemotherapy-surviving ovarian cancer cells secrete fructose, which signals neighbouring tumour cells to become more metastatic in preclinical models. Exogenous fructose at levels typical of sugary drink intake similarly promotes spread. Mechanistically, fructose suppresses cholesterol synthesis, weakening cell–cell adhesion; statins mimic this effect, suggesting potential interaction with chemotherapy responses.

The researchers next aimed to understand why fructose increases cancer cell spread. Through a variety of large-scale analytical techniques, including the use of a CRISPR screen, they discovered that fructose suppresses cholesterol within neighboring cells. Cholesterol is important for cells to stick to each other like a biological glue, so decreased cholesterol allows cells to more easily escape and spread.

The finding that fructose lowers cholesterol production has important clinical implications. Statins, which are taken by 39 million people in the United States, lower cholesterol production.

The team found that statins alone reduced the glue between cells, promoting escape. The research team is now examining whether these medications could be interfering with the effects of chemotherapy—though they stress this isn't a reason for patients to stop taking them.

The researchers are also looking into how this mechanism might extend beyond ovarian cancer.

The chemotherapy-induced senescence-associated secretome promotes cell detachment and metastatic dissemination through metabolic reprogramming, Nature Aging (2026). DOI: 10.1038/s43587-026-01172-5.

New technique pinpoints human DNA inherited from 'ghost' ancestors

Researchers have identified regions of DNA that humans inherited from two unknown ancestors as a result of long-ago interbreeding among modern humans and at least four other archaic human relatives.

While previous research found hints that modern humans had interbred with ancient hominins in addition to now-extinct Neanderthals and Denisovans, the current study pinpoints areas of the genome inherited from these unknown ancestors and establishes a timeline. This was possible using a new technique the researchers developed that leverages hundreds of genomes from modern-day humans to find ancient genealogical relationships.
One of the unknown ancestors, which the researchers refer to as a ghost ancestor, interbred with modern humans in Africa prior to 50,000 years ago, before the most recent migration of Homo sapiens out of Africa into Europe and Asia. These genes comprise around 1% of the genomes of modern humans, comparable to the amount of Neanderthal DNA in the human genome. That unknown hominin lineage split from the modern human lineage around the same time that Neanderthals and Denisovans diverged, about 800,000 years ago, though it interbred with modern humans earlier.

A genealogical method (TRACE) applied to modern human genomes identifies ~2% archaic DNA from two unknown hominin lineages. A “ghost” lineage, diverging ~800,000 years ago, contributed ~0.5–1% of DNA to all humans via gene flow in Africa >50,000 years ago. A “super-archaic” lineage (~1.8 million years old) introgressed into Denisovans, then indirectly into modern humans.

Yulin Zhang et al, Recovering signatures of archaic hominin introgression using ancestral recombination graphs, Science (2026). DOI: 10.1126/science.aef8874. www.science.org/doi/10.1126/science.aef8874

Fewer retractions with open peer-review

Papers with open peer-review reports are less likely to be retracted, says a preprint study. Researchers analysed more than 116,000 papers in Public Library of Science (PLOS) journals and found that among the 40% that opted for publicly available peer-review reports, there were around 100 retractions. There were more than 300 in the closed-review group. It might be that researchers who are confident in their work and who broadly embrace open-science practices are more likely to choose open peer review. In that case, the choice itself could serve as an indicator of article quality.

https://osf.io/preprints/metaarxiv/2b7z5_v1

https://www.nature.com/articles/d41586-026-02282-1?utm_source=Live+...

We overestimate the positive impact of our actions on the climate

Why is recycling waste or switching off the lights sometimes perceived as more effective for the climate than giving up air travel? A team from the University of Geneva (UNIGE) surveyed nearly 3,000 people and identified key psychological mechanisms underlying these misperceptions. Their findings pave the way for more effective communication strategies that encourage behaviors with the greatest potential to reduce greenhouse gas emissions. The study is published in Nature Sustainability.

Carbon competence refers to the ability to accurately assess the impact of different individual actions on greenhouse gas (GHG) emissions. A high level of carbon competence enables people to identify the most effective ways to reduce emissions, both individually and collectively, thereby contributing to climate change mitigation. Yet several studies show that carbon competence remains limited across the general population, including among people who are highly concerned about environmental issues.

As a result, people tend to overestimate the climate impact of relatively modest actions, such as recycling or printing double-sided. While these behaviors are beneficial, their effect on greenhouse gas emissions remains limited. By contrast, people often underestimate the impact of far more effective actions, such as giving up air travel or reducing car use.

Based on a sample of nearly 3,000 participants, a team from UNIGE examined the psychological barriers that explain these misperceptions. The team also tested the effectiveness of three measures designed to improve carbon competence.

They selected 32 individual actions, ranging from low-impact behaviours to those with much greater environmental benefits. They then analyzed how participants estimated the positive impact of these actions when adopted by an average person.

Among the 32 actions studied, the most overestimated low-impact behaviors are, in order: recycling, using recycled materials, purchasing more energy-efficient household appliances, improving home insulation and teleworking. By contrast, the most underestimated high-impact behaviors include giving up air travel, living without a car, switching to an electric vehicle, avoiding long-haul flights and purchasing renewable electricity.

According to the research team, this gap in carbon competence can be explained by two psychological biases: the "availability heuristic," a mental shortcut whereby people judge an action as more effective if it easily comes to mind, and "motivated reasoning," which serves to preserve a positive self-image. "In the first case, the more an action is present in the media or discussed at school, the more easily it comes to mind, and the more likely people are to consider it effective.

In the case of motivated reasoning, individuals tend to believe that the actions they already engage in are more effective because this perception allows them to preserve a positive self-image.

The researchers also show that individuals with strong pro-environmental values tend to be overly optimistic and to overestimate the impact of environmental actions in general. By contrast, those with a stronger capacity to handle numbers tend to provide more accurate estimates and are less influenced by these biases.

Part 1

To correct these misperceptions, the research team tested several interventions. In particular, they assessed the effect of simply providing a list of high-impact actions on participants' estimates of greenhouse gas emissions.

They also examined whether participants could improve their estimates by relying on two indicators: the behavioural cost of an action (the more effort, time or financial resources it requires, the more likely it is to have a high impact) and its social prevalence (the rarer it is within the population, e.g., the U.S., the greater its potential to reduce carbon emissions tends to be).
The challenge is therefore to tailor awareness campaigns to different population profiles—which still need to be defined—in order to better correct these biases and encourage the adoption of climate actions and support for policies that are most effective in reducing greenhouse gas emissions.

The results show that the effectiveness of these interventions varies considerably from one individual to another, particularly depending on their ability to use behavioural cost or social prevalence as a cue of an action's actual impact.

Mario Herberz et al, Psychological barriers to improving carbon competence, Nature Sustainability (2026). DOI: 10.1038/s41893-026-01900-0

Part 2

Hot weather can make pain worse. Here's why
Higher temperatures, particularly ≥32°C, are associated with increased reports of generalized physical pain, especially among middle-aged and lower-income adults. Dehydration, inflammation, blood-pressure changes, stress, disrupted sleep, and reduced activity may amplify pain. Heat-related pain can impair cognition and productivity and may impose substantial economic costs.

original article.

How your conscious and unconscious mind interact
Most brain activity, including autonomic regulation, implicit memory, and rapid affective processing, occurs without awareness but can influence decisions. Conscious processing is more effortful and may become important during novelty or uncertainty. Theories including global workspace and predictive-processing models describe consciousness as integrating or updating information, but remain debated.

Microplastics cross placenta-like barrier and disrupt key pregnancy hormones in new cell model study

Tiny, near-invisible fragments of plastic shed from the products we use every day through sunlight and mechanical wear have been quietly working their way deep into the human body. Microplastics have already been found in the human brain, semen and even the placenta. A recent study investigated how these micro- and nanoplastics (MNPs) might move through the placenta and whether they affect a baby's development.

Since human pregnancy is too complex to study and raises ethical concerns, the team built a miniature lab model that mimics the interaction among a mother, the placenta and the fetus. It combines three types of human cells to represent the different layers of the placental barrier and the fetal adrenal gland. They tested several types of plastics we encounter daily, including PMMA (transparent, glass alternative), PET (used in soda bottles), PVC (used in pipes and packaging) and polystyrene (used in packing materials).

All the types of plastic tested were able to move from the mother's side through the placental barrier to the baby's side. The MNPs didn't just pass through untouched: Some were absorbed into the cells that make up the placenta and the baby's blood vessels. The particles also disrupted levels of hormones such as etiocholanolone and estrogen, which are essential for the mother's health and the baby's development.

Plastics, made from petroleum-derived synthetic or semisynthetic polymers and a cocktail of chemical additives that make them hard, flexible, durable or transparent, have become so pervasive that they now contaminate even the most remote environments. As a result, human exposure to microplastics is inevitable. These tiny plastic particles have been detected in everyday foods and drinks, including salt, seafood, beer and tea, and, in some parts of the world, even in the air.

Part 1

Recent studies have found these plastics in the placenta, amniotic fluid and even a baby's first stool. In general, the placenta, an organ that develops during pregnancy, is responsible for providing oxygen and essential nutrients to the developing fetus while removing waste products and keeping harmful substances at bay. However, researchers are concerned that, because of their extremely small size, MNPs might cross a placenta-like barrier and disrupt hormones.

In this study, the researchers investigated the interaction among a mother, the placenta and a developing baby via a tri-culture model, in which they grew three different types of human cells together to act like a real body system. The model included BeWo cells for the placental barrier, HUVEC cells for the fetal blood vessels and H295R cells for the fetal adrenal glands, which produce hormones such as adrenaline and sex steroids.

To track the particles' journey, the researchers tagged the MNPs with a tiny fluorescent marker and added them to the maternal side of the model. They then watched to see whether the particles could make their way to the fetal side.

They saw that smaller particles moved through the barrier more easily than larger ones. PMMA showed the largest measured transfer, with about 11% reaching the fetal-side chamber after three days.

High-powered 3D microscopes revealed that the plastics weren't just floating around the cells but had actually entered them. No particles were found in the H295R cells, but very small polystyrene particles between 50 nm and 200 nm were found inside both the BeWo and HUVEC cells.
The MNPs also acted as endocrine disruptors. Polystyrene caused a drop in estrogen, a hormone essential for maintaining a healthy pregnancy, and decreased the expression of the CYP17 gene in fetal cells, which is essential for the synthesis of steroid hormones. Almost all the other plastics tested reduced levels of a hormone called etiocholanolone, which sends vital signals to the growing fetus.

These findings warn that various types of microplastics may disrupt hormones during pregnancy and affect placental function. The researchers say more work is needed to understand what this could mean for a baby's development and exactly how these tiny plastic particles disrupt the body's hormone-making machinery.

Jeske van Boxel et al, Effects of different micro- and nanoplastic polymers on steroidogenesis in a feto-placental in vitro model, Molecular and Cellular Endocrinology (2026). DOI: 10.1016/j.mce.2026.112871

Part 2

The brain's electrical signatures shift as tinnitus becomes chronic

Ears are built to listen to sounds from the world around us, but for some people, the sound comes from within. Tinnitus causes people to hear ringing or other noises in one or both ears or in their heads, even when there is no actual sound outside. While tinnitus is frequently associated with hearing loss, a substantial proportion of individuals with clinically normal hearing thresholds still experience it, and the underlying mechanism remains murky to scientists. A recent study investigated how brain network patterns change as tinnitus progresses from a new condition to a long-term chronic one in people with clinically normal hearing.

After recording the electrical activity in the brains of people with acute and chronic tinnitus, the researchers found that the two conditions have very different neurophysiological profiles. People with recent-onset tinnitus showed an imbalance between two important brain networks: the salience network, which detects salient or potentially threatening signals, and the executive control network, which supports attention and decision-making. The brain significantly increases its focus on the former while decreasing the latter's activity. Those with chronic tinnitus showed much more balanced switching between these networks, meaning the brain began to adapt rather than worsen.

Meng-Fang Gong et al, EEG microstates and dynamic functional connectivity reveal stage-specific brain networks in subjective tinnitus, iScience (2026). DOI: 10.1016/j.isci.2026.116995

Researchers uncover hidden mechanism behind congenital heart disease

Congenital heart disease affects approximately two in every 100 newborns globally. But why does it occur? An important part of the answer may lie in a previously unknown mechanism on the surface of our cells. Researchers identified this mechanism in a new study.
They have discovered a new communication system on the exterior of the cell that is crucial for the proper formation of the heart during embryonic development.

The mechanism is located in the primary cilium, a microscopic "antenna" that protrudes from most cells in the body. The cilium's role is to interpret the body's signaling molecules, enabling the cell to determine whether it should divide, move or die, for example.

In the study, the researchers show that three proteins, TAK1, TAB2 and PKA-Cα, function as a signaling hub within the cell's antenna and play a significant role in heart formation.

These proteins act as molecular instructions that tell stem cells when and how to develop into heart muscle cells. However, genetic alterations can disrupt this communication, causing 'antenna defects,' which may lead to congenital heart defects.

The researchers observed effects not only in the heart.

In the study, the rare genetic mutations were identified in patients with so-called syndromic congenital heart disease. Syndromic heart defects are caused by an underlying genetic syndrome that often also leads to defects and associated conditions in other organs.

At the same time, experiments in zebrafish and detailed studies of cilia in other tissues suggested that the mechanism is also important for the development of other organs.

When the ciliary mechanism fails, it typically affects the development of several other organs as well. This may explain why some patients with congenital heart disease also have defects and related conditions affecting the brain, kidneys and skeleton. The mechanism provides a unifying explanation for diseases that we have previously struggled to understand.
The researchers therefore think that the discovery could help improve the understanding and treatment of a wide range of diseases caused by defects in the primary cilium.
This new knowledge may eventually make it easier to identify patients early and develop targeted treatments.

TAK1 operates at the primary cilium in non-canonical TGFB/BMP signaling to control heart development, PLOS Biology (2026). DOI: 10.1371/journal.pbio.3003902

Reducing night lights in cities has ecological effects

Artificial light at night (ALAN) can cause plants to open their buds and sprout leaves earlier in the spring. ALAN can also cause trees to delay changing the colour of their leaves in the fall. These changes can ripple through ecosystems, affecting pollinators, herbivores and insects that lay eggs on plants. ALAN also disorients many migrating birds and other animals.
Some cities have attempted to reduce overall ALAN using techniques such as shielding fixtures, implementing adaptive dimming, reducing light output and choosing warmer colour temperatures.

Urban light-mitigation measures in New York City and Washington, D.C., reduced artificial light at night, particularly blue wavelengths, unlike nearby comparison cities. Reduced lighting was associated with less advanced spring green-up and less delayed autumn leaf coloration, indicating potential mitigation of ALAN-driven shifts in plant phenology.
The authors found that New York City and Washington, D.C., showed a reduction in ALAN after the policies were implemented, especially in the blue band, while Newark and Philadelphia did not.

Plant growth cycles also differed between the cities. ALAN typically advances the start of the growing season and delays the end of the season, but in cities with mitigation policies, the start of the season is several days less advanced than it would otherwise be, as measured by the emergence of new green growth visible in satellite images. Cities with mitigation policies also show a transition to fall colour that is less delayed than in cities without such policies.

According to the researchers, the findings demonstrate the feasibility of using high-resolution satellite images to identify relative differences in ALAN levels and point toward ways in which this tool could support urban ecological research.

Mitigating artificial light at night: effectiveness of policies and implications for plant phenology, PNAS Nexus (2026). DOI: 10.1093/pnasnexus/pgag237 academic.oup.com/pnasnexus/art … /5/8/pgag237/8750162

What's in the dust? Study finds hidden metals in city streets

If you're in an urban area with strong winds, chances are you are breathing in more than a little dust. You could be exposed to metals from cars, construction sites or pesticides used decades ago.

Road dust from cities contained toxic metals, including lead, arsenic, and cadmium, in one-third of samples. Metal concentrations varied with legacy industrial activity, pesticide use, and port operations. Average concentrations remained below health recommendations, but dry conditions may increase exposure.
These dust samples are not from the middle of the highway. They were collected at public spaces, such as parks, schools and downtown areas where residents are likely to be exposed.

Minh Tri Truong et al, Tracing spatial mid-size Eastern U.S. cities road dust pollution: insights from source apportionment and health risk assessment, Environmental Science and Pollution Research (2026). DOI: 10.1007/s11356-026-38034-x

Seaweed-derived coating keeps strawberries fresher than a fridge

Researchers have developed an edible, seaweed-derived coating that can keep strawberries fresh for at least four days at room temperature, outperforming uncoated berries stored in the refrigerator.

An edible agar–zinc–tannic acid coating reduced moisture loss, maintained firmness, and preserved vitamin C and antioxidants in strawberries. Coated berries remained mold-free for ≥4 days at room temperature and ≥6 days refrigerated, exceeding untreated fruit. The coating showed antibacterial activity, no intestinal-cell toxicity, and a lower estimated carbon footprint than refrigeration.
Published in the Journal of Agricultural and Food Chemistry, the findings offer a simple approach for extending produce shelf life and reducing reliance on refrigeration across the supply chain. Maintaining cold temperatures from farm to warehouse to truck to grocery store requires significant energy and infrastructure, especially for fresh produce transported over long distances.
The coating is made from agar, a substance derived from red seaweed that is widely used as a vegan alternative to gelatin and as a thickener in foods like puddings and jellies. On its own, agar forms a thick gel. But when researchers combined it with zinc, an essential nutrient, and tannic acid, a naturally occurring plant compound found in grapes and tea, the material transformed—self-assembling into tiny microparticles that create a thin protective layer around the fruit.

When dipped in the solution, strawberries, grapes and apple slices emerged with a thin, edible coating that dried completely clear.

The breakthrough could help address one of the food industry's most persistent challenges: reducing food waste.

Ivy Chiu et al, Self-Assembled Metal–Phenolic–Agar Microparticle-Derived Coatings Enable Scalable and Sustainable Fresh Produce Preservation, Journal of Agricultural and Food Chemistry (2026). DOI: 10.1021/acs.jafc.6c06531

Forest floors reveal overlooked seed dispersers: Slugs, beetles and crabs spread viable seeds

A review by researchers has revealed that tiny ground-dwelling invertebrates are overlooked but important seed dispersers, challenging the traditional focus on birds and mammals and offering new insights into forest regeneration and biodiversity conservation.
A synthesis of 43 publications documented viable seed dispersal by at least 51 invertebrate species across 186 plant taxa. Slugs, earthworms, beetles, crickets and crabs can deposit seeds in favourable microsites, sometimes enhancing germination. This short-range dispersal may support regeneration where vertebrate dispersers decline.
The findings highlight an important but underappreciated pathway of seed dispersal that may help sustain plant regeneration, especially in fragmented or degraded ecosystems where larger animal dispersers are declining.

Si-Chong Chen et al, Invertebrate endozoochory: An overlooked pathway of seed dispersal, Trends in Plant Science (2026). DOI: 10.1016/j.tplants.2026.06.004

Like Zika, Oropouche virus can also affect the brains of unborn babies
Oropouche virus infected human neural stem cells and brain organoids, reducing proliferation, disrupting tissue organization, and increasing inflammatory and cell-death pathways. Historical and recent strains caused similar effects, suggesting inherent neurodevelopmental risk. Organoid results support biological plausibility for congenital abnormalities but do not establish pregnancy outcomes.

Gabrielle Brum et al, Microcephaly-like phenotype triggered by novel reassortant and prototypic Oropouche virus strains in brain organoids, eBioMedicine (2026). DOI: 10.1016/j.ebiom.2026.106408

original article.

Social determinants of health linked to more fatalistic cancer beliefs

New research shows that greater cumulative exposure to adverse social determinants of health is associated with beliefs that cancer is unavoidable or a death sentence. This can influence whether people choose to be screened for common cancers or seek treatment after a diagnosis.
Among 1,400 respondents, greater cumulative exposure to adverse social determinants of health was associated with stronger fatalistic beliefs that cancer is unavoidable or inevitably fatal. Poverty, housing insecurity, lack of insurance, discrimination, and social isolation may contribute to reduced screening follow-up and treatment engagement.

Jaitri Joshi et al, Effect of social stressors and social determinants of health (SDOH) on cancer beliefs: Analysis of a cancer center catchment area, Journal of Psychosocial Oncology (2026). DOI: 10.1080/07347332.2026.2666535

Brain scans reveal tau buildup in late-onset psychosis

Hallucinations and delusions that appear for the first time in midlife or later are often treated as psychiatric symptoms whose biological cause is unclear. The study, published in Molecular Psychiatry suggests that, for many patients, these symptoms may be linked to brain changes also seen in dementia.
Among 37 people with psychosis onset after age 40, 65% had tau PET positivity and 35% had amyloid positivity, versus 15% and 2% of 47 healthy controls. Tau patterns included amyloid-positive Alzheimer-type and amyloid-negative forms; greater parietal tau in amyloid-positive patients correlated with poorer executive function.
Using positron emission tomography, or PET, the research team found that about 65% of patients who developed psychosis after age 40 showed abnormal buildup of tau, a protein associated with Alzheimer's disease and other forms of dementia. By comparison, tau PET positivity was seen in about 15% of healthy older controls. The study also found amyloid PET positivity in 35% of patients, compared with 2% of controls.

The findings provide the first in vivo evidence that diverse tau-related brain changes may be involved in late-onset psychosis. Until now, earlier epidemiological and postmortem studies had suggested a possible relationship between late-life psychotic symptoms and neurodegeneration, but direct evidence in the living brain had been limited.

Patients can show the same symptoms, such as hallucinations or delusions, but their clinical course and response to treatment can be very different.

Manabu Kubota et al, High prevalence of tau pathologies in late-onset psychosis: A PET study, Molecular Psychiatry (2026). DOI: 10.1038/s41380-026-03749-3

Act now on microplastic risks, businesses urged

Microplastics are pervasive in products, supply chains and ecosystems, with emerging evidence of human-health and environmental harm. Plastic production exceeds 450 million tonnes annually and may nearly triple without intervention. Businesses face increasing regulatory, reputational and litigation risks, requiring monitoring, reporting and reduction of primary and secondary microplastic pollution.

As the science and engineering behind industrial monitoring advance, businesses will be expected to track the level of microplastics in their products and control secondary microplastic pollution."

The white paper highlights many actions businesses can take now to proactively manage risks related to microplastic exposure.

Increased monitoring and transparent reporting, circular business models, recycled-content products and materials innovation can build trust and generate efficiencies in the face of consumer concern.

Businesses that communicate credibly about their microplastic footprints and align marketing practices with actual reduction commitments will be better placed to build durable brand equity.

The question is not whether supply chains including plastics will change but how quickly, and who will lead it.

Martina Linnenluecke et al, Microplastics: Preparing for Australia's Next Regulatory Shift, University of Technology Sydney (2026). DOI: 10.71741/4pyxmbnjaq.32583060

Sunlight-powered setup generates quantum entanglement

Quantum entanglement is crucial for applications such as secure communication, ultra-precise sensing and high-performance computation.

Today's quantum technologies rely on energy-intensive lasers, raising concerns that scaling them up could further increase energy demands. In new work, researchers have demonstrated that quantum entanglement between photons can be generated directly from sunlight, offering a potential alternative.
In Optica, the researchers report that the entanglement they achieved using sunlight was comparable to laser-based approaches after accounting for differences in the bandwidth of the input light.
This technology could one day enable satellites to create secure encryption keys using the sunlight already abundant in space, reducing the need for onboard lasers and much of the supporting hardware.
Sunlight-driven entanglement generation could also provide the crucial ingredient needed to scale up quantum computing without adding to the energy burden.
Researchers conducted an outdoor experiment at MPL. Using quantum state tomography to characterize the resulting quantum state, they found that the entanglement generated from sunlight was about 94% similar to a perfectly entangled state. Additionally, the photons produced during the experiment were correlated in a way that violated Bell's inequality, indicating that the correlation has no classical explanation and must be a result of quantum entanglement.

Now that the proof-of-principle demonstration is complete, the researchers are working toward a field-deployable product by making improvements in brightness and the quality of entanglement.

Cheng Li et al, Generating quantum entanglement from sunlight, Optica (2026). DOI: 10.1364/optica.601797

Common blood storage bags may leach microplastics into stored blood

In the 1970s, PVC bags revolutionized the storage of blood and blood products for transfusion. Disposable plastic (PVC) bags replaced reusable glass bottles, making the process more convenient and safer from contamination. Over half a century later, the same disposable plastic bags are creating a new kind of problem because of microplastics (MPs). These tiny particles, less than 5 millimeters in size, are formed by the polymer degradation of plastic materials as they break down and age.

In a recent study published in Environment & Health, researchers from China investigated how microplastics are released from medical storage bags into the blood and preservation fluids used for transfusions.

They found PVC microplastics not only in blood-preservation fluids but also in stored blood samples, with microscopy showing that some particles attached to red blood cells. Their levels rose substantially during storage, increasing by roughly seven- to 20-fold over 30 days compared with freshly opened blood bags in the tested systems.

Storage conditions also played a role in the amount of plastic being shed. Microplastic levels increased with higher storage temperatures, and the bags were shaken to simulate transportation.

Microplastics do not belong anywhere on this planet, but they have made their way to every corner of Earth. There is growing evidence that these microscopic plastic particles have invaded many tissues and organs throughout the human body, including the lungs, liver, placenta, blood and blood vessels.

Studies have already documented that microplastics leach from everyday disposable medical gear, including IV infusion sets, syringes, IV bags and even the tubing used in kidney dialysis. For instance, IV infusion bags alone can shed up to 5,455 plastic particles into every litre of fluid.

Blood bags are typically made of PVC.

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High-tech techniques, including Raman spectroscopy and scanning electron microscopy, revealed that tiny, irregular plastic fragments floating in blood storage solutions were PVC particles shed from the bags. Raman spectroscopy identified the particles' chemical fingerprints, while scanning electron microscopy captured microscopic changes in blood transfusion bags. The microscopy also showed that plastic particles attached directly to the outer membranes of stored red blood cells.

Tests revealed that storing blood bags at room temperature caused significantly more plastic to break off than storing them at 4 °C did. Simulated transport placed additional physical stress on the bags, causing the plastic to crack and peel and releasing significantly more particles into the liquid than in bags that remained still.

When healthy red blood cells were exposed to these microplastic-containing fluids for 30 days, their intracellular ATP—the chemical energy that keeps them alive and maintains their shape—gradually declined. Some cells also lost their disk shape and became spiky over time.

The researchers also identified a potential solution—ultrafiltration, which passes the storage liquid through extremely fine filters before it is added to blood. It removed 78%–85% of the microplastics. With most of the particles filtered out, red blood cells retained their normal shape, maintained their energy levels and were protected from shape changes.

The findings provide direct evidence that microplastics contaminate blood storage systems and can affect the quality of stored blood.

Ning Li et al, Microplastics Released from Blood Storage Bags: A Hidden Contribution to Red Blood Cell Storage Lesions, Environment & Health (2026). DOI: 10.1021/envhealth.6c00110

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Decades of data reveal who's most at risk of snakebites and how to prevent them

Snakebites aren't random but are predictably correlated with a variety of environmental and socioeconomic factors, according to a study published Aug. 6, 2026, in the journal PLOS Neglected Tropical Diseases .
Snakebite is considered a neglected tropical disease, responsible for as many as 1.4 million deaths per year and disproportionately affecting rural populations in low- and middle-income countries. However, limited information is available on the environmental and socioeconomic factors influencing snakebite incidence.
Across 142 studies (>200,000 cases), nearly 70% of snakebites occurred during outdoor work, especially farming and construction. Risk increased during rainy seasons and evening hours and was associated with poverty-related factors including inadequate footwear, insecure housing, and limited health education. Prevention should complement antivenom through workplace safety, housing, rural development, and education.
In the study, researchers synthesized data from 142 studies documenting more than 200,000 snakebites worldwide dating to the 1800s. The review found that snakebite is strongly related to occupation, with nearly 70% of cases occurring during outdoor work such as farming and construction. It is also related to timing, with most cases occurring during rainy seasons and during the evening and early-night hours. Snakebite cases were also correlated with factors related to poverty, such as lack of adequate footwear, poorly secured homes and limited access to health education.
These results highlight areas where targeted policies might significantly reduce the risk of snakebite, including workplace safety, rural development, housing improvement and health education. The authors note that the dataset is limited because it is mostly focused on South Asia and has inconsistent reporting of variables involved in each snakebite incident.
The authors say, "The scoping review told us something we needed to hear: limiting ourselves to antivenom will never address the behavioural and environmental factors that put people at risk in the first place. The inverse care law is particularly relevant to snakebite; the people most exposed to snakes—because of where they live and the work they do—are also the people furthest from a hospital that can treat them. You cannot fix that gap with antivenom alone; antivenom sits at the wrong end of the problem."

Behavioural and environmental risk factors associated with snake–human conflict: A scoping systematic review, PLOS Neglected Tropical Diseases (2026). DOI: 10.1371/journal.pntd.0014505

Why do people's pupils dilate when they're surprised?

When people encounter new information that challenges their expectations, their pupils are likely to dilate. This physiological response is a sign that something is happening in the brain to help the person adapt to the new situation, according to a new study.
Surprising events elicit activity in the part of the brain known as the locus coeruleus, which is the primary source of norepinephrine, the chemical messenger that drives the body's fight-or-flight stress response. This activity is correlated with a change in pupil diameter as well as with specific brain waves measured by electroencephalography (EEG).
The researchers found in their experiments that surprising colours—those that didn't match predictions—elicited pupil dilation and amplified certain brain waves. They also found that these measurements were related to reductions in bias and adjustments in learning: When images looked like what the participants thought they were going to see, participants were biased toward their expectations. Yet pupil and brain measurements showed that participants were also able to learn from unexpected results and adjust their subsequent predictions accordingly.

In surprising situations, the researchers found, the norepinephrine spike is almost like a refresh mechanism—it's a sign the brain is adjusting to new information that changes expectations.
The brain holds on to some mental context, and then when it recognizes that you're in a new situation, you replace that context. Changes in pupil diameter, as well as specific EEG readings, are external markers showing that the brain is loading that new context. This both limits the effect that the previous context has on perception and also provides a clean slate, unencumbered by previous expectations, thereby allowing one to learn faster.

Tiantian Li et al, Fluctuations in arousal reflect latent state transitions that facilitate behavioural optimization, Nature Human Behaviour (2026). DOI: 10.1038/s41562-026-02533-1

Scientists discover a brain cycle that may explain why learning eventually stalls

For many who have struggled to learn a new language or instrument, it's a familiar pattern: They make rapid progress at first, then hit a plateau that no amount of diligent practice or concentration seems to break.

Biologists identified a physical mechanism in the brain that may help explain why such plateaus occur: A scaffold-like structure surrounding brain cells, called the extracellular matrix, helps control when the brain can change during learning. In the auditory cortex, this matrix loosens within hours of a practice session and rebuilds itself within about a day—a cycle that lets the learning from a session take hold before the next one. As a skill is mastered, the rebuilding cycle fades and eventually stops altogether, as if the brain has decided that learning is finished and it's time to protect what's been gained.
In the adult auditory cortex, extracellular matrix loosens within hours of training and rebuilds by about 24 hours, supporting learning and consolidation. This remodelling declines and ceases as skills are mastered. Enzymatic matrix disruption impaired learning and degraded retained performance, indicating roles in both plasticity and stability.

Caras, Melissa L., Experience-dependent modulation of extracellular matrix integrity supports perceptual skill learning and memory, Proceedings of the National Academy of Sciences (2026). DOI: 10.1073/pnas.2601801123

Dry needling is different from acupuncture. So what's involved? And is it effective?
Dry needling inserts solid needles into painful muscle areas, unlike acupuncture’s traditional point selection. It may provide short-term relief for neck, shoulder, and low-back pain but appears no more effective than exercise or other treatments, with uncertain placebo-independent benefit. Minor adverse effects are common; rare serious harms include pneumothorax and infection.

 original article.

Taxi drivers rarely die of Alzheimer's. How complex mental maps and spatial reasoning protect your brain
Taxi and ambulance drivers had among the lowest Alzheimer’s mortality rates, potentially because continual navigation engages hippocampal spatial-memory systems. Intensive navigation is associated with hippocampal structural changes, and complex environments correlate with lower Alzheimer’s risk. Whether screen-based spatial work provides similar protection remains untested.

original article.

Is Morbi's Mysterious 'Dancing Water' : Mystery Finally Solved

An 18-foot-deep farm well in Virparda village of Gujarat's Morbi district has gone viral after producing continuous, tide-like waves since August 2. While the unusual sight sparked fears of seismic activity, experts have ruled out any connection to an earthquake. The phenomenon is reportedly caused by heavy upstream rainfall rapidly recharging underground aquifers. The increased groundwater pressure forces trapped air out of rock pores, creating rhythmic bubbling and oscillating waves inside the well. Trapped air beneath the water may be causing the ripples.

Morbi District Collector Swapnil Khare has ordered a scientific assessment.

And the above explanation is what science gives!

Mushroom behind 'tiny people' hallucinations identified

People in communities thousands of miles apart have described the same strange experience after eating a mysterious mushroom: vivid visions of tiny humans moving through and interacting with the physical world around them. Known as Lilliputian hallucinations—a reference to the 6-inch-tall inhabitants of "Gulliver's Travels"—the phenomenon has long been thought to stem from cultural influences rather than biology.

A new study suggests otherwise.

Using DNA sequencing, researchers confirmed that a single mushroom species, Lanmaoa asiatica, is responsible for these hallucinations in Southwest China and the northern Philippines.

Three completely independent cultures have reported the same specific type of hallucination, and two cases are attributed to the same DNA-verified mushroom species. That indicates that these bizarre psychological effects aren't cultural manifestations or coincidences—they must have a shared underlying chemical and neurological basis.
People pretty much always report seeing dozens or hundreds of little people, about 3 to 30 centimeters tall (1 to 12 inches), in incredible detail, like they're actually there. About 90% of people describe them as little elves or clowns or other fairy-like figures dressed in colorful clothes.

What's fascinating is they interact with the physical world using the laws of physics that govern us. They're not walking through walls or anything. They're falling off the edge of tables, walking around objects. One person told me that while they were eating soup, the little people were jumping off their spoon into the bowl and swimming around. As they scooped a bite, the little people remained in their mouth.

They also discovered that the mushroom contains none of the psychoactive compounds known to science, including psilocybin. Instead, the findings point to an entirely new hallucinogenic compound that could offer new insights into neurological disease and how the brain shapes perception and consciousness.
However, Lilliputian hallucinations predate this mushroom. There are myths about tiny people in pretty much every culture's folklore. People have also reported these hallucinations from alcohol withdrawal, dementia, macular degeneration and other neurological conditions. So, it seems like this phenomenon is fundamental to how the human mind and brain work.
That's what makes this mushroom different—it reliably produces this effect, whereas those conditions very rarely trigger it.

Colin Domnauer et al, Phylogenomic systematics of Lanmaoa (Boletaceae) reveals cryptic diversity, resolves global evolutionary relationships, and suggests a novel psychoactive lineage, Mycologia (2026). DOI: 10.1080/00275514.2026.2670968

Early-life stress leaves a 'scar' inside brain cells

More than half of the world's children are exposed to early-life stress from abuse, household dysfunction such as violence or drug use, or other traumatic experiences. Accumulation of four or more such experiences can trigger much higher risks for long-term mental and physical health challenges in adulthood.

Experiencing severe stress during childhood can make a person more vulnerable to anxiety, depression and other mood disorders when faced with hardships as an adult. Researchers have uncovered how trauma early in life can leave a lasting effect on the brain.
Scientists already knew that stress early in life changes the activity of genes in the brain. In a new study, the research team discovered that this is due to alterations in how brain cells package DNA, leaving the brain's genetic stress response vulnerable to being easily turned on and reducing tolerance to stress.
This new finding reveals a physical scar left by trauma experienced during development inside brain cells, providing scientists with a concrete biological target to develop new treatments and interventions.
The researchers set out to understand how trauma during early development physically changes the brain to make it more sensitive to stress later in life. They focused on a region of the brain called the ventral tegmental area, where brain cells that produce dopamine—a chemical messenger—are responsible for processing important things in the environment, including rewards and adversity.
When these brain cells are activated abnormally, which can happen in response to stress, they disrupt how the brain processes rewards, leaving individuals vulnerable to anxiety and depression.

Within dopamine-producing neurons, the researchers zoomed in on the epigenome, a set of molecular tags that direct the cell's machinery to turn genes on and off, which in turn affects cells' activity.

Inside cells, DNA is coiled like a slinky. The DNA coils are wrapped around histone proteins that help determine how tightly or loosely the coil is wound.

When the genetic slinky is compressed, its genes are turned off. As the DNA slinky stretches and opens, genes are more easily accessible to be turned on.

The researchers found that an enzyme called SETD7 was more abundant in the dopamine neurons of young mice that had experienced stress compared with mice reared in a typical environment. SETD7 helps place a chemical tag—H3K4me1—on the genetic slinky, marking the structure for uncoiling, which in turn makes the cell more reactive to everything going on in the environment.
The researchers then artificially boosted SETD7 in young, stress-free mice. Even without early-life stress, these mice grew up with a stretched-open DNA structure in their dopamine-producing brain cells, making it easier to turn on the genes that respond to stress. Such mice had a lower tolerance for stress in adulthood.

The researchers found that, as adults, mice whose SETD7 levels had been boosted when they were young had more reactive dopamine neurons and more anxious behavior compared with mice with normal levels of SETD7 throughout their lives.
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Conversely, when the researchers blocked the SETD7 enzyme from adding too much of the H3K4me1 tag after early-life stress, the slinky remained closed, shielding mice from becoming hypersensitive to stress later in life. Despite experiencing both early-life and adult stress, mice with their SETD7 levels dampened were able to remain as social and exploratory as unstressed mice, and their dopamine neurons were active at normal levels.

There are currently no treatments for what early-life stress does to the brain, partially because we have not had a clear picture of what molecular mechanisms to target.
This work is exciting because it reveals a clear mechanism and also helps explain why the impact of stress is both latent and broad. Additionally, if we can step in with supportive care, therapy or social resources to buffer children during those sensitive windows of development, we may be able to protect the epigenome—preventing the genetic slinky from locking into an open position and perhaps giving the developing brain a chance to build natural resilience.

Kim HJJ, et al. Early-life stress alters H3K4me1 in VTA to prime stress sensitivity, Neuron (2026).

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Meta says its AI model hacked another company, adding to worries about bots going rogue
A Meta AI model gained unintended internet access during cybersecurity testing and exploited a third-party vulnerability; the incident is under investigation. Related tests reported unsanctioned online actions by AI agents under reduced safeguards, highlighting containment needs for cyber-capability evaluations.
source: News agencies

Brain activity reveals two stages of visual processing before conscious perception
EEG decoding indicated early occipital activity (~200 ms) encoding individual visual offsets and their temporal order without conscious separation. Later central-parietal activity (~450 ms) represented the integrated percept and behavioural reports, consistent with sequential unconscious encoding followed by conscious visual integration.

Maëlan Q. Menétrey et al, Sequential neural dynamics underlie unconscious integration and conscious perception of visual stimuli, PLOS Biology (2026). DOI: 10.1371/journal.pbio.3003894

How to keep the Earth habitable when the sun dies

In a billion years, the sun will start to run out of fuel and expand to become a red giant. Earth's oceans and atmosphere will be burned away, and Earth will likely be engulfed by the sun altogether. The sun will then shrink to a dim white dwarf, and all light and warmth in the solar system will diminish. In a hundred trillion years, the galaxy's ability to produce stars will be exhausted, and everything will turn dark. However, there's a way for life to escape this fate—and what's more, we wouldn't need to leave the planet to do it. We just need to build some safeguards.

A proposed long-term strategy uses a sunshade, artificial fusion power from gas giants, and orbital adjustment to protect Earth during solar evolution. Antimatter-driven core heating could sustain tectonics, while directed mass beams could alter Solar System motion. The concept remains highly speculative and requires extreme resources.

Gabriel Harry, Retaining Earth's Habitability Beyond the Life of the Sun, Journal of the British Interplanetary Society (2026). DOI: 10.59332/jbis-079-07-0230. On arXiv: DOI: 10.48550/arxiv.2607.13084.

Brain turns listening inward during REM sleep, EEG recordings suggest
Across wakefulness to phasic REM sleep, EEG responses to external sounds progressively decreased, while responses to heartbeats were preserved or strengthened in 25 participants. This indicates a selective shift from external to internal sensory processing rather than global sensory suppression. An audio–cardiac response index may help characterize altered consciousness.

Sensory processing reallocation from auditory to cardiac signals in REM sleep, Current Biology (2026). DOI: 10.1016/j.cub.2026.07.024

A baby's lungs may begin responding to vitamin D sooner than once thought
Many pregnant women around the world are low in vitamin D. This is a very common issue that has prompted inquiries over the years. It is believed that low levels in pregnant women can cause harm to foetal development. Currently, scientists are asking a controversial question. What if the key time for vitamin D effects is before a woman knows that she's pregnant? By week 16 of pregnancy, the structural makeup of their developing airways—that is, the blueprint of breathing—is complete.
This timing begs an interesting question. According to a recent review published in Nutrients, it is not the amount of the nutrient that is important. Rather, it is the timing of the availability of vitamin D.

In short, this is the most important factor that would help us understand childhood asthma. New research suggests that there should be more focus on supplementation during the initial weeks, even before conception. The bulk of the early lung development generally occurs by the time clinical trials first start taking place.
All earlier vitamin D studies were conducted after conception, never before.
A 2025 Cochrane review affirms this trend. The research found that giving a high dose of vitamin D during pregnancy probably protects against wheezing in childhood, though it probably makes "little to no difference" to doctor-diagnosed asthma. Giving more vitamin D to pregnant women lowered wheezing in toddlers on average but did not convincingly prevent an asthma diagnosis.

Notably, these studies pointed out that all these trials started quite late and usually measured vitamin D only once. The researchers say that these designs yield only "weak evidence against the periconceptional hypothesis because they were not designed to test it."
The main takeaway is this: taking vitamin D before pregnancy might matter more than taking huge doses afterwards.

In the early weeks, when the fundamental branches of the lungs are established, it is likely that vitamin D benefits airway programming the most.

The review noted that none of the large trials, in fact, tested giving vitamin D pre-conception or in the first trimester. If timing matters, starting the supplements after 20 weeks may have been too late. The researchers argue that focusing on a higher dose in late pregnancy won't uncover an asthma-prevention effect if it was needed earlier.

Looking to the future, the researchers call for new trials that begin several weeks before conception and continue through early pregnancy. They also suggest measuring vitamin D repeatedly (not just once) and following children longer, with objective lung tests. Such studies could identify if a "vitamin D vacuum" in early pregnancy leaves lungs more vulnerable to asthma.

Oana Raluca Temneanu et al, Timing over Dose: Maternal Vitamin D, Periconceptional Window, and Early-Life Respiratory Programming, Nutrients (2026). DOI: 10.3390/nu18142333

People carrying a rare gene variant have naturally low body fat, healthier livers and better blood sugar levels

There are people around us who can maintain their weight despite what they eat. This has led scientists to believe that a person's metabolism and tendency toward obesity may have genetic factors behind them. A large study sequencing the genomes of more than 1 million people across three continents, found genetic clues that could drive this tendency. The findings are published in Nature.
They used exome sequencing to identify the specific parts of DNA responsible for building proteins, looking for rare gene variants and their associations with the ratio of triglycerides to HDL cholesterol (TG) in the blood (TG: HDL), a marker linked to several heart and metabolic risk factors and diseases.

They pinpointed 59 genes associated with this marker that help regulate how the body balances, stores and burns energy. One of the major players is the FNIP1 (folliculin-interacting protein 1) gene, which essentially acts as a brake on energy expenditure, stopping cells from burning too much energy.

About 1 in 7,000 people carry a rare, naturally occurring genetic mutation that switches off one copy of the FNIP1 gene. This anomaly appeared to come with its share of metabolic advantages, including significant protection against obesity and liver fat, a more favourable distribution of body fat and around 60% lower odds of cardiometabolic disease, among others.
In this study, the researchers built a massive data set that allowed them to identify meaningful patterns in genetic variants associated with healthier metabolism. The team collected genetic data and health records from 1,032,116 people across 11 study groups spanning North America, Europe and Asia.

To read the participants' DNA, they focused specifically on the exome, the 1% of our DNA that contains the instructions for making proteins. Then they looked at TG: HDL in their blood and matched those results with advanced diagnostic data. Using a powerful software program called REGENIE, they searched for genetic variants associated with higher or lower TG: HDL.

The study showed that a simple blood measurement like the TG: HDL ratio, which is the ratio of triglycerides to HDL, the "good" cholesterol, can act as a highly accurate indicator of full-body metabolic health. A higher TG: HDL ratio was strongly associated with excess body fat, dangerous fat buildup around vital organs, insulin resistance and higher blood pressure.

Fifty-nine independent genes were found to directly shape how the body balances energy, stores fat and manages metabolism. Nearly all of them operate in the liver and fat tissue, the body's core metabolic hubs. Of these, 23 genes—close to 40%—already had drugs targeting them, either approved or being tested in clinical trials.

They found that carriers of the rare FNIP1 gene variant had a very healthy metabolic profile. Compared with noncarriers, they showed lower levels of blood fats, including triglycerides and bad cholesterol; lower body weight and body-fat percentage; and a healthier distribution of body fat. They also had lower blood sugar and significantly less fat stored in the liver.
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The researchers then tested the effect in mice by disabling the FNIP1 pathway in the liver. Even when fed a high-fat, high-sugar junk-food diet for up to 30 weeks, these mice were strongly protected against weight gain and fat buildup in the liver.

The results identify FNIP1 as a key player in how the human body manages energy and metabolic biomarkers. Researchers are hopeful that blocking it therapeutically could eventually be used to tackle heart disease, obesity and diabetes.

George Hindy et al, FNIP1 variants are associated with favourable metabolism in 1 million humans, Nature (2026). DOI: 10.1038/s41586-026-10864-2

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