Science, Art, Litt, Science based Art & Science Communication
JAI VIGNAN
All about Science - to remove misconceptions and encourage scientific temper
Communicating science to the common people
'To make them see the world differently through the beautiful lense of science'
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Latest Activity: 17 hours ago
WE LOVE SCIENCE HERE BECAUSE IT IS A MANY SPLENDOURED THING
THIS IS A WAR ZONE WHERE SCIENCE FIGHTS WITH NONSENSE AND WINS
“The greatest enemy of knowledge is not ignorance, it is the illusion of knowledge.”
"Being a scientist is a state of mind, not a profession!"
"Science, when it's done right, can yield amazing things".
The Reach of Scientific Research From Labs to Laymen
The aim of science is not only to open a door to infinite knowledge and wisdom but to set a limit to infinite error.
"Knowledge is a Superpower but the irony is you cannot get enough of it with ever increasing data base unless you try to keep up with it constantly and in the right way!" The best education comes from learning from people who know what they are exactly talking about.
Science is this glorious adventure into the unknown, the opportunity to discover things that nobody knew before. And that’s just an experience that’s not to be missed. But it’s also a motivated effort to try to help humankind. And maybe that’s just by increasing human knowledge—because that’s a way to make us a nobler species.
If you are scientifically literate the world looks very different to you.
We do science and science communication not because they are easy but because they are difficult!
“Science is not a subject you studied in school. It’s life. We 're brought into existence by it!"
Links to some important articles :
1. Interactive science series...
a. how-to-do-research-and-write-research-papers-part 13
b. Some Qs people asked me on science and my replies to them...
Part 6, part-10, part-11, part-12, part 14 , part- 8,
part- 1, part-2, part-4, part-5, part-16, part-17, part-18 , part-19 , part-20
part-21 , part-22, part-23, part-24, part-25, part-26, part-27 , part-28
part-29, part-30, part-31, part-32, part-33, part-34, part-35, part-36, part-37,
part-38, part-40, part-41, part-42, part-43, part-44, part-45, part-46, part-47
Part 48, part49, Critical thinking -part 50 , part -51, part-52, part-53
part-54, part-55, part-57, part-58, part-59, part-60, part-61, part-62, part-63
part 64, part-65, part-66, part-67, part-68, part 69, part-70 part-71, part-73 ...
.......306
BP variations during pregnancy part-72
who is responsible for the gender of their children - a man or a woman -part-56
c. some-questions-people-asked-me-on-science-based-on-my-art-and-poems -part-7
d. science-s-rules-are-unyielding-they-will-not-be-bent-for-anybody-part-3-
e. debate-between-scientists-and-people-who-practice-and-propagate-pseudo-science - part -9
f. why astrology is pseudo-science part 15
g. How Science is demolishing patriarchal ideas - part-39
2. in-defence-of-mangalyaan-why-even-developing-countries-like-india need space research programmes
3. Science communication series:
a. science-communication - part 1
b. how-scienitsts-should-communicate-with-laymen - part 2
c. main-challenges-of-science-communication-and-how-to-overcome-them - part 3
d. the-importance-of-science-communication-through-art- part 4
e. why-science-communication-is-geting worse - part 5
f. why-science-journalism-is-not-taken-seriously-in-this-part-of-the-world - part 6
g. blogs-the-best-bet-to-communicate-science-by-scientists- part 7
h. why-it-is-difficult-for-scientists-to-debate-controversial-issues - part 8
i. science-writers-and-communicators-where-are-you - part 9
j. shooting-the-messengers-for-a-different-reason-for-conveying-the- part 10
k. why-is-science-journalism-different-from-other-forms-of-journalism - part 11
l. golden-rules-of-science-communication- Part 12
m. science-writers-should-develop-a-broader-view-to-put-things-in-th - part 13
n. an-informed-patient-is-the-most-cooperative-one -part 14
o. the-risks-scientists-will-have-to-face-while-communicating-science - part 15
p. the-most-difficult-part-of-science-communication - part 16
q. clarity-on-who-you-are-writing-for-is-important-before-sitting-to write a science story - part 17
r. science-communicators-get-thick-skinned-to-communicate-science-without-any-bias - part 18
s. is-post-truth-another-name-for-science-communication-failure?
t. why-is-it-difficult-for-scientists-to-have-high-eqs
u. art-and-literature-as-effective-aids-in-science-communication-and teaching
v.* some-qs-people-asked-me-on-science communication-and-my-replies-to-them
** qs-people-asked-me-on-science-and-my-replies-to-them-part-173
w. why-motivated-perception-influences-your-understanding-of-science
x. science-communication-in-uncertain-times
y. sci-com: why-keep-a-dog-and-bark-yourself
z. How to deal with sci com dilemmas?
A+. sci-com-what-makes-a-story-news-worthy-in-science
B+. is-a-perfect-language-important-in-writing-science-stories
C+. sci-com-how-much-entertainment-is-too-much-while-communicating-sc
D+. sci-com-why-can-t-everybody-understand-science-in-the-same-way
E+. how-to-successfully-negotiate-the-science-communication-maze
4. Health related topics:
a. why-antibiotic-resistance-is-increasing-and-how-scientists-are-tr
b. what-might-happen-when-you-take-lots-of-medicines
c. know-your-cesarean-facts-ladies
d. right-facts-about-menstruation
e. answer-to-the-question-why-on-big-c
f. how-scientists-are-identifying-new-preventive-measures-and-cures-
g. what-if-little-creatures-high-jack-your-brain-and-try-to-control-
h. who-knows-better?
k. can-rust-from-old-drinking-water-pipes-cause-health-problems
l. pvc-and-cpvc-pipes-should-not-be-used-for-drinking-water-supply
m. melioidosis
o. desensitization-and-transplant-success-story
p. do-you-think-the-medicines-you-are-taking-are-perfectly-alright-then revisit your position!
q. swine-flu-the-difficlulties-we-still-face-while-tackling-the-outb
r. dump-this-useless-information-into-a-garbage-bin-if-you-really-care about evidence based medicine
s. don-t-ignore-these-head-injuries
u. allergic- agony-caused-by-caterpillars-and-moths
General science:
a.why-do-water-bodies-suddenly-change-colour
b. don-t-knock-down-your-own-life-line
c. the-most-menacing-animal-in-the-world
d. how-exo-planets-are-detected
e. the-importance-of-earth-s-magnetic-field
f. saving-tigers-from-extinction-is-still-a-travail
g. the-importance-of-snakes-in-our-eco-systems
h. understanding-reverse-osmosis
i. the-importance-of-microbiomes
j. crispr-cas9-gene-editing-technique-a-boon-to-fixing-defective-gen
k. biomimicry-a-solution-to-some-of-our-problems
5. the-dilemmas-scientists-face
6. why-we-get-contradictory-reports-in-science
7. be-alert-pseudo-science-and-anti-science-are-on-prowl
8. science-will-answer-your-questions-and-solve-your-problems
9. how-science-debunks-baseless-beliefs
10. climate-science-and-its-relevance
11. the-road-to-a-healthy-life
12. relative-truth-about-gm-crops-and-foods
13. intuition-based-work-is-bad-science
14. how-science-explains-near-death-experiences
15. just-studies-are-different-from-thorough-scientific-research
16. lab-scientists-versus-internet-scientists
17. can-you-challenge-science?
18. the-myth-of-ritual-working
19.science-and-superstitions-how-rational-thinking-can-make-you-work-better
20. comets-are-not-harmful-or-bad-omens-so-enjoy-the-clestial-shows
21. explanation-of-mysterious-lights-during-earthquakes
22. science-can-tell-what-constitutes-the-beauty-of-a-rose
23. what-lessons-can-science-learn-from-tragedies-like-these
24. the-specific-traits-of-a-scientific-mind
25. science-and-the-paranormal
26. are-these-inventions-and-discoveries-really-accidental-and-intuitive like the journalists say?
27. how-the-brain-of-a-polymath-copes-with-all-the-things-it-does
28. how-to-make-scientific-research-in-india-a-success-story
29. getting-rid-of-plastic-the-natural-way
30. why-some-interesting-things-happen-in-nature
31. real-life-stories-that-proves-how-science-helps-you
32. Science and trust series:
a. how-to-trust-science-stories-a-guide-for-common-man
b. trust-in-science-what-makes-people-waver
c. standing-up-for-science-showing-reasons-why-science-should-be-trusted
You will find the entire list of discussions here: http://kkartlab.in/group/some-science/forum
( Please go through the comments section below to find scientific research reports posted on a daily basis and watch videos based on science)
Get interactive...
Please contact us if you want us to add any information or scientific explanation on any topic that interests you. We will try our level best to give you the right information.
Our mail ID: kkartlabin@gmail.com
Started by Dr. Krishna Kumari Challa. Last reply by Dr. Krishna Kumari Challa 17 hours ago. 1 Reply 0 Likes
Why do some birds dance?Cockatoos perform 30 distinct dance moves and may combine them in unique waysCaptive cockatoos have at least 30 different dance moves in their repertoire, including…Continue
Started by Dr. Krishna Kumari Challa. Last reply by Dr. Krishna Kumari Challa yesterday. 2 Replies 0 Likes
From fabricated research to paid authorships and citations, organized scientific fraud is on the rise, according to a new study.By combining large-scale data analysis of scientific literature with…Continue
Started by Dr. Krishna Kumari Challa. Last reply by Dr. Krishna Kumari Challa on Tuesday. 1 Reply 0 Likes
Your phone is covered in germs: A tech expert explains how to clean it without doing damageWe wash our hands, sanitize shopping trolleys and wipe down cafe tables. But what about our phones? We touch…Continue
Started by Dr. Krishna Kumari Challa. Last reply by Dr. Krishna Kumari Challa on Sunday. 1 Reply 0 Likes
Q: Why do you feel very sleepy after lunch?Krishna: I don’t! During the day, I mean. Because I work the whole day and don’t sit idle, my brain will always be in a highly alert state.And I eat my…Continue
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Why do some birds dance?
Captive cockatoos have at least 30 different dance moves in their repertoire, including headbanging and body rolls, according to a new study.
The moves, of which 17 are newly identified, may be performed with or without music.
Several species of parrot have been anecdotally observed dancing to music in captivity. Dancing results from complex brain processes including imitation, learning and synchronized, rhythmic movement. Spontaneous dancing in time to music has only been reported in humans and parrots, although some wild birds also display rhythmic movements as part of their courtship displays.
Researchers analyzed 45 videos posted on social media that showed cockatoos (Cacatuidae) dancing. They identified a total of 30 distinct dance movements—17 of which had not previously been described scientifically. These newly identified dance moves included headbanging, sidesteps and body rolls.
The researchers found that some birds also performed their own individual dance moves, often by combining several of the movements in unique ways. Closely related species did not display more similar dances, and each species had a unique top 10 most common dance moves.
A team of theoretical researchers used thermal effective theory to demonstrate that quantum entanglement follows universal rules across all dimensions. Their study was published online in Physical Review Letters.
In classical physics, two particles that are far apart behave independently. However, in quantum physics, two particles can exhibit strong correlations regardless of the distance between them. This quantum correlation is known as quantum entanglement.
Quantum entanglement is a fundamental phenomenon underlying quantum technologies such as quantum computation and quantum communication, and understanding its structure is important both theoretically and practically.
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One of the key measures used to quantify quantum entanglement is the Rényi entropy. Rényi entropy quantifies the complexity of quantum states and the distribution of information, and plays a crucial role in the classification of quantum states and in assessing the feasibility of simulating quantum many-body systems.
Moreover, Rényi entropy serves as a powerful tool in theoretical investigations of the black hole information loss problem, and frequently appears in the context of quantum gravity.
Most studies to date have been limited to (1+1)-dimensional systems, or 1 spatial dimension plus time dimension. In higher dimensions, analyzing the structure of quantum entanglement becomes significantly more difficult.
A group of researchers has shown the universal features of quantum entanglement structures in higher dimensions by applying theoretical techniques developed in the field of particle physics to quantum information theory.
The research team focused on the thermal effective theory, which has recently led to major advances in the analysis of higher-dimensional theories in particle physics. This is a theoretical framework designed to extract universal behavior from complex systems, based on the idea that observable quantities can often be characterized by only a small number of parameters.
By introducing this framework into quantum information theory, the team analyzed the behavior of Rényi entropy in higher-dimensional quantum systems.
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Yuya Kusuki et al, Universality of Rényi Entropy in Conformal Field Theory, Physical Review Letters (2025). DOI: 10.1103/fsg7-bs7q
**
AI is a relatively new tool, and despite its rapid deployment in nearly every aspect of our lives, researchers are still trying to figure out how its "personality traits" arise and how to control them. Large learning models (LLMs) use chatbots or "assistants" to interface with users, and some of these assistants have exhibited troubling behaviors recently, like praising evil dictators, using blackmail or displaying sycophantic behaviors with users. Considering how much these LLMs have already been integrated into our society, it is no surprise that researchers are trying to find ways to weed out undesirable behaviors.
Anthropic, the AI company and creator of the LLM Claude, recently released a paper on the arXiv preprint server discussing their new approach to reining in these undesirable traits in LLMs. In their method, they identify patterns of activity within an AI model's neural network—referred to as "persona vectors"—that control its character traits. Anthropic says these persona vectors are somewhat analogous to parts of the brain that "light up" when a person experiences a certain feeling or does a particular activity.
Anthropic's researchers used two open-source LLMs, Qwen 2.5-7B-Instruct and Llama-3.1-8B-Instruct, to test whether they could remove or manipulate these persona vectors to control the behaviors of the LLMs. Their study focuses on three traits: evil, sycophancy and hallucination (the LLM's propensity to make up information). Traits must be given a name and an explicit description for the vectors to be properly identified.
In their method, a technique called "steering" can be used to control behaviors. They write, "When we steer the model with the 'evil' persona vector, we start to see it talking about unethical acts; when we steer with 'sycophancy,' it sucks up to the user; and when we steer with 'hallucination,' it starts to make up information. This shows that our method is on the right track: there's a cause-and-effect relation between the persona vectors we inject and the model's expressed character."
However, they found that when they made these changes after training, the model loses some of its intelligence. But there was a workaround—the team found that inducing the bad behaviors during training allowed the LLMs to integrate better behavior without reducing their usefulness. Furthermore, they found that they can monitor and predict persona shifts during deployment and training and flag problematic training data that is more likely to produce unwanted traits, even before fine-tuning the model.
Runjin Chen et al, Persona Vectors: Monitoring and Controlling Character Traits in Language Models, arXiv (2025). DOI: 10.48550/arxiv.2507.21509
Researchers have shown that a low-oxygen environment—similar to the thin air found at Mount Everest base camp—can protect the brain and restore movement in mice with Parkinson's-like disease.
The new research, in Nature Neuroscience, suggests that cellular dysfunction in Parkinson's leads to the accumulation of excess oxygen molecules in the brain, which then fuel neurodegeneration—and that reducing oxygen intake could help prevent or even reverse Parkinson's symptoms.
The fact that researchers actually saw some reversal of neurological damage is really exciting. It tells us that there is a window during which some neurons are dysfunctional but not yet dead—and that we can restore their function if we intervene early enough.
The results raise the possibility of an entirely new paradigm for addressing Parkinson's disease.
Parkinson's disease, which affects more than 10 million people worldwide, causes the progressive loss of neurons in the brain, leading to tremors and slowed movements.
Neurons affected by Parkinson's also gradually accumulate toxic protein clumps called Lewy bodies. Some biochemical evidence has suggested that these clumps interfere with the function of mitochondria—the tiny powerhouses of the cell that Mootha knew were altered in other diseases that could be treated with hypoxia.
Moreover, anecdotally, people with Parkinson's seem to fare better at high altitudes. And long-term smokers—who have elevated levels of carbon monoxide, leading to less oxygen in tissues—also appear to have a lower risk of developing Parkinson's.
Based on this evidence, researchers became very interested in the effect of hypoxia on Parkinson's disease.
The results of their experiments were striking. Three months after receiving α-synuclein protein injections, the mice breathing normal air had high levels of Lewy bodies, dead neurons, and severe movement problems. Mice that had breathed low-oxygen air from the start didn't lose any neurons and showed no signs of movement problems, despite developing abundant Lewy bodies.
The findings show that hypoxia wasn't stopping the formation of Lewy bodies but was protecting neurons from the damaging effects of these protein clumps—potentially suggesting a new mode of treating Parkinson's without targeting α-synuclein or Lewy bodies.
What's more, when hypoxia was introduced six weeks after the injection, when symptoms were already appearing, it still worked. The mice's motor skills rebounded, their anxiety-like behaviors faded, and the loss of neurons in the brain stopped.
However, more work is needed before the findings can be directly used to treat Parkinson's.
Marutani, E et al. Hypoxia ameliorates neurodegeneration and movement disorder in a mouse model of Parkinson's disease, Nature Neuroscience (2025). DOI: 10.1038/s41593-025-02010-4
Before the first polio vaccine was developed in 1955, the disease—spread mostly from person to person, through contaminated water and via fecal particles—was among the world's most feared, paralyzing hundreds of thousands of children annually. People avoided crowded places during epidemics, and hospital wards filled with children encased in iron lungs after the virus immobilized their breathing muscles.
Polio is mainly spread when people are exposed to water infected with the virus. In countries with poor sanitation, children often become infected when they come into contact with contaminated waste.
WHO says that as long as a single child remains infected, kids everywhere are at risk.
Eradication demands near-perfection—zero polio cases and immunizing more than 95% of children.
But public health leaders and former WHO staffers say campaign efforts are far from perfect, and many question the oral vaccine.
The oral vaccine—proven to be safe and effective—has been given to more than 3 billion children. But there are some extremely rare side effects: Scientists estimate that for every 2.7 million first doses given, one child will be paralyzed by the live polio virus in the vaccine.
In even rarer instances, the live virus can mutate into a form capable of starting new outbreaks among unimmunized people where vaccination rates are low.
Worldwide, several hundred vaccine-derived cases have been reported annually since at least 2021, with at least 98 this year.
Most public health experts agree the oral vaccine should be pulled as soon as possible. But they acknowledge there simply isn't enough injectable vaccine—which uses no live virus and doesn't come with the risks of the oral vaccine—to wipe out polio alone. The injectable vaccine also is more expensive and requires more training to administer.
https://medicalxpress.com/news/2025-08-world-polio-fake-imperfect-v...
Part 2
**
For the past decade, health care workers have traveled door to door in southeastern Pakistan, pleading with parents to allow children to be vaccinated against polio as part of a global campaign to wipe out the paralytic disease. She hears their demands and fears. Some are practical—families need basics like food and water more than vaccines. Others are simply unfounded—the oral doses are meant to sterilize their kids.
Amid rampant misinformation and immense pressure for the campaign to succeed, some managers have instructed workers to falsely mark children as immunized. And the vaccines, which must be kept cold, aren't always stored correctly.
In many places, their work is not done with honesty. The result: Polio raised its ugly head again.
The World Health Organization and partners embarked on their polio campaign in 1988 with the bold goal of eradication—a feat seen only once for human diseases, with smallpox in 1980. They came close several times, including in 2021, when just five cases of the natural virus were reported in Pakistan and Afghanistan. But since then, cases rebounded, hitting 99 last year, and officials have missed at least six self-imposed eradication deadlines.
Afghanistan and Pakistan remain the only countries where transmission of polio—which is highly infectious, affects mainly children under 5, and can cause irreversible paralysis within hours—has never been interrupted. The worldwide campaign has focused most of its attention and funding there for the past decade.
But in its quest to eliminate the disease, the Global Polio Eradication Initiative has been derailed by mismanagement and what insiders describe as blind allegiance to an outdated strategy and a problematic oral vaccine, according to workers, polio experts and internal materials.
Officials have falsified vaccination records, selected unqualified people to dole out drops, failed to send out teams during mass campaigns, and dismissed concerns about the oral vaccine sparking outbreaks, according to documents shared with some news agencies.
The documents flagged multiple cases of falsified vaccination records, health workers being replaced by untrained relatives and workers improperly administering vaccines.
Part 1
A team of biologists has discovered that nighttime caffeine consumption can increase impulsive behavior, potentially leading to reckless actions.
The study, published in iScience, examined how nighttime caffeine intake affects inhibition and impulsivity in fruit flies.
Drosophila melanogaster, the fruit fly species used in the study, is a powerful model to study complex behaviors due to its genetic and neural parallels with humans. Caffeine is the most widely consumed psychoactive substance in the world.
Interestingly, caffeine consumed by the flies during the daytime did not lead to the same reckless flying, the team said.
The team warns that the findings could have negative implications for shift workers, health care and military personnel who consume coffee at night, particularly females.
Erick Benjamin Saldes et al, Nighttime caffeine intake increases motor impulsivity, iScience (2025). DOI: 10.1016/j.isci.2025.113197
A new preclinical study has found exposure to nanoplastics may contribute to the rapid progression of Alzheimer's disease and subsequent spread from the brain to other key organs such as the liver, heart and gut.
The research, "Cerebral to Systemic Representations of Alzheimer's Pathogenesis Stimulated by Polystyrene Nanoplastics," is published in the journal Environment & Health
The study investigated how environmental-level polystyrene nanoplastic exposure influences the progression of Alzheimer's disease from the brain to other parts of the body. Studies in mice revealed that nanoplastic-induced neurological damage is not confined within the brain, but expands systemically through the gut–liver–brain axis.
In mice exposed to polystyrene nanoplastics, Alzheimer's-like symptoms were shown to stimulate 'microglia' immune cell activation in the brain, leading to neuroinflammation and neurodegeneration. This in turn led to peripheral health implications beyond the brain, including fatty liver disease, abnormal build-up of fat and gut microbiota imbalance.
Humans are involuntarily exposed to plastics through inhalation, dermal contact and the consumption of contaminated food and water, and plastic particles have been detected in human lungs, bloodstream and, very recently, in the human brain.
This, however, is the first study to show how rapidly nanoplastics can evolve from the brain to other parts of the body.
Yue Wang et al, Cerebral to Systemic Representations of Alzheimer's Pathogenesis Stimulated by Polystyrene Nanoplastics, Environment & Health (2025). DOI: 10.1021/envhealth.5c00160
For decades, medical professionals debated whether a common antiviral medication used to treat flu in children caused neuropsychiatric events or if the infection itself was the culprit.
Now researchers at Monroe Carell Jr. Children's Hospital at Vanderbilt have debunked a long-standing theory about oseltamivir, known as Tamiflu.
According to the study, published in JAMA Neurology, oseltamivir treatment during flu episodes was associated with a reduced risk of serious neuropsychiatric events, such as seizures, altered mental status and hallucination.
These findings demonstrated what many pediatricians have long suspected, that the flu, not the flu treatment, is associated with neuropsychiatric events.
"In fact, oseltamivir treatment seems to prevent neuropsychiatric events rather than cause them."
Scientists have documented a notable case of antibiotic resistance evolving within a critically ill patient during treatment for an E. coli bloodstream infection, providing genomic evidence of how drug resistance can emerge in real time.
This new study published in the Journal of Medical Microbiology, details the rapid evolution of resistance in an E. coli strain exposed to piperacillin/tazobactam (TZP), a first-line treatment for serious bacterial infections that pairs an antibiotic with a compound that inhibits beta-lactamase enzymes, a widespread antibiotic resistance gene.
While the initial infection appeared treatable, the bacteria quickly developed a mechanism to escape the drug's effects, not by acquiring new resistance genes, but by amplifying one it already carried, overcoming the effects of the resistance inhibitor.
This is a striking example of resistance evolving under antibiotic pressure.
The researchers identified a tenfold increase in copies of a key resistance gene within the bacterial isolate, leading to a 32-fold increase in the level of antibiotic required to kill the bacteria, ultimately causing the treatment to fail, and all within the course of a single patient's illness.
The research team, which included genomic scientists, microbiologists and clinicians used high-resolution whole-genome sequencing to confirm the genetic changes.
The amplified resistance gene in E. coli, named blaTEM-1, produces a beta-lactamase enzyme that breaks down the antibiotic piperacillin. Although the TZP drug combination is meant to inhibit these enzymes, the sheer volume produced following gene duplication overwhelmed its protective effect, allowing the infection to persist. Further lab experiments confirmed that exposure to TZP led E. coli to generate even more copies of the gene.
This form of "within-patient evolution" presents a major diagnostic challenge. Routine resistance tests may underestimate the risk of treatment failure if they don't detect bacteria capable of rapidly increasing enzyme production under antibiotic pressure.
The study also highlights that 40% of new antibiotic candidates in the pipeline are beta-lactamase inhibitor combinations like TZP, raising critical concerns for drug developers and frontline clinicians alike.
This study underscores why relying on static resistance profiles can be misleading.
The findings underscore the need for greater investment in diagnostics and surveillance tools that can detect dynamic, hard-to-spot resistance mechanisms before they undermine treatment.
Alice J. Fraser et al, A high-resolution genomic and phenotypic analysis of resistance evolution of an Escherichia coli strain from a critically unwell patient treated with piperacillin/tazobactam, Journal of Medical Microbiology (2025). DOI: 10.1099/jmm.0.002018
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